07 August 2012
Gold alloys contain gold, copper and other metals that results in a strong effective filling, crown or bridge.
Gold alloys are primarily used for inlays, onlays, crowns and fixed bridges.
There are two form of gold fillings, cast gold fillings(gold inlay and onlay) made with 14 or 18 kt gold and gold foil made with pure 24 kt gold.
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TYPES:
1.GOLD INLAY
Gold inlay is basically gold filling which fits within the contours of tooth. The gold inlay involves the occlusal and proximal surface of posterior tooth and may cap one or more but not all of the cusps.
Gold inlay is custom crafted in dental laboratory to precisely fit tooth so it takes two appointments to restore tooth with an inlay.
INDICATION:
1. Replace over amalgam restoration.
2. When proximal surface caries is extensive.
3. Gold inlay restorations for class-1, class-4, class-5, and class-6 cavity.
2. GOLD ONLAY
Gold onlay involves the proximal surface of a posterior tooth and caps all of the cusps.
A gold onlay covers and protects the biting surface of a tooth that is severely damaged and not possible with dental filling.
INDICATION:
1. In posterior teeth after root canal treatment.
2. As a splinting to improve stability of the teeth.
3. Partial coverage of posterior teeth where significant loss of coronal dentine.
ADVANTAGE:
1. Strength (can withstand occlusal forces)
2. Long lasting life (at least 10 to 15 years)
3. Naturally beautiful
4. Gold is pliable.
5. Gold is versatile.
DISADVANTAGES:
1. Expensive
2. Galvanic shock
3. More visit
07 August 2012 by Dr.Sunil Patel · 0
Glass ionomer cement (GIC) is a dental restorative material used in dentistry for filling teeth and luting cements. These materials are based on silicate glass powder and polyalkenoic acid.
TYPES:
Glass ionomer are classified into five types.
1. Conventional glass ionomer cements.
2. Resin modified glass ionomer cements.
3. Hybrid ionomer cements. (Also known as dual cured glass ionomer cement).
4. Tri-cure glass ionomer cements.
5. Metal reinforced glass ionomer cements.
INDICATION:
1. Type-2 glass ionomer for restorations.
2. Type-3 glass ionomer for liners and bases.
3. Type-4 glass ionomer for fissure sealants.
4. Type-5 glass ionomer for core build up.
5. Intermediate Restorations.
6. Restorations for decidious teeth.
7. Adhesive cavity liners(sandwich technique)
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ADVANTAGE:
- Inherent adhesion to tooth structure
- High retention rate
- Little shrinkage and good marginal seal
- Fluoride release and caries inhibition
- Biocompatible
- Minimal cavity preparation
DISADVANTAGE:
- Brittle
- Soluble
- Abrasive
- Water sensitive during setting phase
- Less aesthetic than composite
CONTRAINDICATION:
- Aesthetic requirement
- Area of high abrasion
- Area of high occlusal load
by Dr.Sunil Patel · 0
Dental composite resins are types of synthetic resins which are used in dentistry as restorative material or adhesives.
Composite resins are most commonly composed of BISGMA (Bisphenol Glycidyl Methacrylate).
Now UDMA (urethane dimethacrylate) and TEGDMA (Triethylene glycol dimethacrylate) are also being used.
Fillers used are quartz, silica, tricalcium phosphate, zirconium dioxide. They provide strength, hardness, rigidity to the material.
Coupling agent binds the resin matrix to the filler particles. Organic salines are commonly used coupling agents.
Coloring agents used are aluminium oxide, titanium dioxide.
Composite are divided into types depending upon the size, amount and composition of the inorganic filler.
TYPES:
- Conventional
- Microfilled
- Nanofilled
- Flowable composite
- Laser curing composite
- Packable composite
- Antibacterial composite
Placement of composite requires an etchant and bonding agent.
-On the bases of how they can be used the composite fillings are divided into two types
Direct dental composite
Direct dental composite placed by dentist in clinical one visit.
Composites are available in many shades so according to color of tooth shade is selected to give best esthetic results through single shade, dual shade and multilayering technique.
Before putting the resin in the cavity the area treated with an agent that is called as the etching process.
After etching and bond is applied which is cured by light then composite is placed and is cured with light.
Direct dental composites can be used for
- Filling gaps (diastemas) between teeth using a shell-like veneer.
- Minor reshaping of teeth.
- Partial crowns on single teeth.
INDIRECT DENTAL COMPOSITE
This type of composite is cured outside the mouth.
Indirect composites can have higher filler levels and are cured for longer times
As result they have higher levels and depths of cure then direct composite.
Indirect dental composites can be used for
- Filling cavities in teeth as fillings, inlays and onlays.
- Filling gaps between teeth using a shell-like veneer.
- Reshaping of teeth.
- Full or partial crowns on single teeth.
- Bridges.
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by Dr.Sunil Patel · 0
Dental amalgam is the most commonly used dental restorative material used for dental fillings.
Fillings made with amalgam are also known as silver fillings.
Amalgam consists of combination of metals, these include silver, mercury, tin, copper and small amounts of zinc, iridium or palladium with development of tooth colored materials to restore teeth, amalgam is used less often than in the past.
INDICATION:
Amalgum restorations for class-1 cavity, class-2 cavity, class-3 cavity, class-4 cavity and class-5 cavity.
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ADVANTAGE:
- Long lasting and strong, with stand the forces of chewing.
- Less costly than the other material
DISADVANTAGE:
- amalgam doesn’t match with natural color of tooth.
- amalgam doesn’t bond with natural tooth.
- Requirement of cavity preparation to keep filling in place.
by Dr.Sunil Patel · 0
DEFINITION:“The periodontal pocket defined as a pathological deepened gingival sulcus.” There is also a loss of bone and disorganization of periodontal ligament.
Pocketing is the end result of inflammation and infection that causes the loss of tissue attachment to the teeth, one common consequence of periodontal disease.
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GINGIVAL POCKET (False or Relative)
This is formed by gingival enlargement without destruction of the underlying periodontal tissue. The sulcus is deepened because of increased bulk of gingival.
PERIODONTAL POCKET (True)
This occurs with destruction of the supporting periodontal tissues. Progressive pocket depending leads to destruction of the supporting periodontal tissues and loosening and exfoliation of the teeth.
There are two types of periodontal pockets:
SUPRABONY (supracrestal or supraalveolar) in which the bottom of the pocket is coronal to underlying alveolar bone.
INFRABONY (intrabony, subcrestal or intraalveolar) in which the bottom of the pocket is apical to the level of the adjacent alveolar bone. Lateral pocket wall lies between the tooth surface and alveolar bone.
Pockets can involve one, two, or more tooth surfaces and can be different depths and types on different surfaces of the same tooth and on approximating surface of the same interdental space.
SIGNS AND SYMPTOMS
- bluish red
- thickened marginal gingival
- bluish red vertical zone from the gingival margin to alveolar mucosa.
- gingival bleeding and suppuration
- tooth mobility
- distema formation
Symptoms such as
-localized pain or pain deep in the bone
-pocket depths and extent is determine by careful probing of the gingival margin along each tooth surface.
Pathogenesis:
Periodontal pockets are caused by microorganisms and their products which produce pathologic tissue changes that lead to deepening of the gingival sulcus.
Bacterial invasion:
Bacterial invasion of the apical and lateral areas of the pocket wall.
Filaments,rods,coccoid organisms with predominant gram-negative cell walls have been found in interalveolar spaces of the epithelium.
Bacteria invade the intercellular space under exfoliating epithelial cells but they are also found between deeper epithelial cells and accumulating on the basement lamina and invade the sub epithelial connective tissue.
POCKET CONTENTS:
Periodontal pockets contain debris consisting periapically of microorganisms and their products (enzymes, endotoxins and other metabolic products)
Gingival fluid, food remnants, salivary mucin, desquamated epithelial cells and leukocytes plaque covered calculus usually projects from the tooth surface.
Purulent exudates if present consists of living, degenerated and necrotic leukocytes, living and dead bacteria, serum and scant amount of fibrin.
Extensive pus formation may occur in shallow pockets
Deep pockets may exhibit little or no pus.
Repair, resection, regeneration and extraction.
TREATMENT:
1.Maintain good oral hygiene
2.Scaling & Root planning
3.Laser deep pocket cleaning
4.Gingival curettage
5.Gingivectomy
6.periodontal flap surgery
a) Papilla preservation flap
b) Sulcular incision flap
c) Modified widman flap
d) Undisplaced flap
e) Apically displaced flap with bone contouring
7.Resective osseous surgery
8.Reconstructive osseous surgery
9.tooth extraction or partial tooth extraction
(hemisection or root resection)
by Dr.Sunil Patel · 0
Abscesses of the periodontium are localized acute bacterial infections classified primarily based on location.
Types
There are four types of abscesses associated with the periodontal tissues:-
1. Gingival abscesses
2. Periodontal abscesses
3. Pericoronal abscesses
4. Combined periodontal/endodontic abscesses
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LATERAL PERIODONTAL ABSCESS
Definition: “A periodontal abscess is a localized, purulent infection involves a greater dimension of gum tissues extending apically and adjacent to periodontal pocket such lesion may contribute to destruction of the periodontal ligament and alveolar bone.
Periodontal abscesses can be acute or chronic
Causes of lateral periodontal abscesses
- Continued irritation by calculus, food debris, deposition of foreign body in the interdental spaces
- lateral periodontal abscess is commonly associated to supra and infra bony pockets as they are very hard to clean.
- External root resorption
- cracked tooth
- perforation of lateral wall of tooth during root canal therapy possible cause.
SIGNS AND SYMPTOMS:
- mild, moderate to severe pain
- Gums are reddened, swollen and pus may come out an application of pressure
- tooth may show mobility, tenderness and slight elevation
- fever
- lymph node tenderness is common
- mouth opening may be difficult
TREATMENT:
- Incision and drainage
- Analgesic for eliminate pain
- Antibiotics for eliminate infection
- maintain oral hygiene
- scaling, gingivectomy if necessary
- examination if needed
by Dr.Sunil Patel · 0
Definition: periodontitis is the most common type of periodontal disease and results from extention of the inflammatory process initiated in the gingival to supporting periodontal tissues.
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Etiology of periodontitis
- dental plaque
- Material Alba
- Dental calculus
- Microorganisms
- food debris
- poor oral hygiene
- dental stains
- Iatrogenic factors
- Faulty fixed or Removable prosthesis
- Food impaction
- Unreplaced missing teeth
- Malocclusion
- Mouth breathing
- Tobacco chewing
- Thumb sucking
- Cigarette smoking
- Toothbrush trauma
- chemical irritation
- bruxism
RISK FACTOR
- vit-c
- gingivitis
- Heredity
- poor oral health habits
- Tobacco use
- Diabetes
- older age
- Decreased immunity such as that occurring with leukemia or HIV-AIDS
- Poor nutrition
- certain medication
- Hormonal changes such as related to pregnancy
- ill fitting dental restorations.
PERIODONTITIS
Classified according to rate of progression
1) slowly progressive periodontitis
2)Rapidly progressive periodontitis
-Classified according to age at onset
1)Adult onset periodontitis
2)early onset periodontitis
a) Prepubertal periodontitis.
b) Juvenile periodontitis.
OTHER FORMS OF PERIODONTITIS
1) Aggressive periodontitis
2) Necrotizing ulcerative periodontitis
3) Refractory periodontitis
4) Periodontitis as manifestation of systemic disease
A) Associated with hematological disorders
1. Acquired neutropenia
2. Leukaemias
3. Others
B) Associated with genetic disorders
1. Familial and cyclic neutropenia
2. Down-syndrome
3. Leukocyte adhesion deficiency syndromes.
4.papillon lefevres syndrome
5. chediak-Higashisyndrome
6. Histocytosis syndrome
7. Glycogen storage disease
8. Infantile genetic agranulocytosis
9. Cohen syndrome
10.Ehlers-Danlos syndrome(type 4 and type 8)
11.Hypophosphatasia
12.Other
5)Combined periodontic endodontic lesion
6)Abscess of periodontitis
a)Gingival abscess.
b)Periodontal abscess.
c)peri coronal abscess.
SYMPTOMS OF PERIODONTITIS
- Red swollen or tender gums
- Bleeding gums during tooth brushing
- Halitosis or bad breath
- Gums that have palled always from teeth or gingival recession
- pus formation between teeth and gums
- Loosening or separating teeth
- Deep pockets between teeth and gums.
Periodontitis has been linked to increased inflammation in body such as indicated by raised levels of c-reactive protein and interleukin 6. It is through this linked to increased risk of stroke, myocardial infarction and atherosclerosis.
Diagnosis of periodontitis.
- Oral examination by dentist or dental hygienist.
- By x-rays
- Periodontal probing procedure
- Examination of periodontal pocket
Radiographic changes in periodontitis
Normally interdental septa
- The interdental septum normally presents a thin radio opaque border adjacent to and at the crest of periodontal ligament this border is referred to as laminadura
- Radiographically continuous white line but really it is perforated by numerous small foramina and traversed by blood vessels, lymphatic’s and nerve pass between periodontal ligament and bone.
- changes in angulations of x-rays beam produce considerable variations in its appearance.
AFTER PERIODONTITIS RADIOGRAPHIC CHANGES
- Fuzziness and break in the continuity of laminadura at the mesial and distal aspect of crest of interdental septum.
- Wedge shaped radiolucent area is formed at the mesial or distal aspect of crest of septal bone.
- Finger like radiolucent projections extend from crest into the septum & height is reduced.
- Height of interdental septum is progressively reduced by extention of inflammation and resorption of bone.
- Radioopaque horizontal line across the roots.
# TREATMENT PLAN OF PERIODONTITIS
There are several ways to treat periodontitis depending on its severity, etiology, symptoms and risk factors.
NONSURGICAL TREATMENTS
if periodontitis is not in advanced treatment can include less invasive procedure including
SCALING
Scaling removes plaque and food debris from tooth surfaces and beneath gums. It performed by ultrasonic scaler device.
ROOTPLANNING
Root planning smoothes the root surfaces and present further develop of plaque and calculus.
ANTIBIOTICS:
Topical or oral antibiotics to help control bacterial infection
NSAIDS : To reduce inflammation.
SURGICAL TREATMENTS:
If periodontitis may in advanced then surgical procedure may require such as
FLAP SURGERY
-In this procedure makes small incision in gums so that section of gum tissue is pull back exposing root surface for scaling and rootplaning.periodontitis may be causes bone loss, the underlying bone may be recontoured before the gum tissue
Is sutured back in place. The procedure is under local anesthesia.
SOFTT TISSUE GRAFTS:
If gum tissue lose in periodontal disease, and gumline recedes, making teeth appear longer than normal, then damaged tissue replaced by removing small
This procedure reduces gum recession, cover exposure of root surface, and give aesthetically pleasing appearance.
BONE GRAFTING:
This procedure is performed when periodontitis has destroyed bone surrounding tooth root surface. The graft may be small part of own bone or synthetic bone or donated. This bone graft helps prevent tooth bone loss by holding tooth in proper
Place. It also serve s base for regrowth of natural bone. Bone grafting may be performed during a technique called guided tissue regeneration.
GUIDED TISSUE REGENERATION
This allows the regrowth of bone that was destroying by bacterial infection. In this procedure a special biocompatible material places between existing bone and tooth. This stimulates growth of healthy bone and tissue.
PREVENTION:
- Flossing daily
- Brushing after meals every day.
- Balanced and healthy diet.
by Dr.Sunil Patel · 0
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